Vitamin D Deficiency

Vitamin D deficiency is a condition, in which the phosphate and calcium blood level decrease,  causing bone diseases due to lack of phosphate and calcium that is available to keep a healthy bone. Such condition is called osteomalacia in adults and rickets in children.

Deficiency of Vitamin D may be resulted from either a lack of Vitamin D in the person’s diet or inadequate exposure to sun. Vitamin D deficiency at the time of pregnancy can form osteomalacia in the women and rickets in their newborn. Because human milk does not consists of large vitamin D amount, breastfed infant can progress rickets, even in the tropic areas, if he or she is shielded from the sunlight. Vitamin-D-Deficiency

Vitamin D deficiency can happen in elderly because of lower Vitamin D in the skin production when exposed to sun. Few rare types of rickets, resulted from Vitamin D impaired metabolism are hereditary. Tetany (spasm of the muscles) resulted from a low level of calcium, can be the 1st rickets sign in infant. An older infant can be slow to crawl and sit, and the spaces closing between the fontanelles (skull bones) can be delayed. Children between the ages of one and four can walk slowly, experience defected spine curve, as well as knock-knees and bow legs. For adolescents and older children, walking is painful.

The pelvic bone flattening in adolescent girls can result in the birth canal to narrow. In adults, the calcium loss from bones, especially in the legs, pelvis and spine, result in fracture and weakness. Vitamin D deficiency diagnosis depends on the low Vitamin D by-products, phosphate and calcium blood levels. Osteomalacia and rickets may be treated by taking Vitamin D orally in daily doses 5 times the advised daily allowance for two or three weeks.  Specific hereditary rickets forms generally improve when treated with hormone containing Vitamin D.

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